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This directly indicated increased tolerance to virus infection stress could be an effect of co-involvement of GSH and NPR1 which was observed in tolerant interaction of tobacco with other types of pathogens (Zechmann and Mller, 2008)

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In particular, the Lys8, Trp207 and Lys210 residues showed structural differences from their corresponding residues compared with the oxidized and GSH-bound structures and rearrangement of these residues is related to the movements necessary for the interactions involved in AsA binding (Fig